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Par freypo freypo le jeudi, mars 1 , Amphetamine USAN, abbreviated from alpha-methylphenethylamine or amfetamine INN is a psychostimulant drug of the phenethylamine class that produces increased wakefulness and focus in association with decreased fatigue and appetite. Brand names of medications that contain, or metabolize into, amphetamine include Adderall, Dexedrine, Dextrostat, Desoxyn,[2] ProCentra, and Vyvanse, as well as Benzedrine in the past. The drug is also used recreationally and as a performance enhancer. Recreational users of amphetamine have coined numerous street names for amphetamine, such as "speed". Withdrawal symptoms of amphetamine consist primarily of mental fatigue, mental depression and increased appetite. Symptoms may last for days with occasional use and weeks or months with chronic use, with severity dependent on the length of time and the amount of amphetamine used. Withdrawal symptoms may also include anxiety, agitation, excessive sleep, vivid or lucid dreams, deep REM sleep and suicidal ideation.

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In manic-depressives, a shift towards the manic phase may occur; should the patient enter a manic phase amitriptyline should be discontinued. As described for other psychotropics, amitriptyline may modify insulin and glucose responses calling for adjustment of the antidiabetic therapy in diabetic patients; in.

Addition the depressive illness itself may affect patients' glucose balance. Hyperpyrexia has been reported with tricyclic antidepressants when administered with anticholinergic or with neuroleptic medications, especially in hot weather. After prolonged administration, abrupt cessation of therapy may produce withdrawal symptoms such as headache, malaise, insomnia and irritability. Amitriptyline should be used with caution in patients receiving SSRIs see sections 4. Amitriptyline for enuresis should not be combined with an anticholinergic drug.

Suicidal thoughts and behaviours may also develop during early treatment with antidepressants for disorders other than depression; the same precautions observed when treating patients with. Depression should therefore be followed when treating patients with enuresis. Long-term safety data in children and adolescents concerning growth, maturation and cognitive and behavioural development are not available see section 4.

Opioids possessing a high protein-binding profile may have an increased free-drug level in patients with liver insufficiency because of their decreased. Production of alphaacid glycoprotein and albumin. Hence, high protein-binding properties may lead to toxicity. The liver is the major site of biotransformation from parent opioid compounds to active or inactive metabolites. Unfortunately, neither of these tools nor other endogenous markers are able to provide any assessment of hepatic clearance; therefore, an alternative approach in.

Determining drug dosing is needed in the hepatic impairment patient population.

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